Patholgy p1 mcqs

 cell injury

1 What is the Type of necrosis seen in Infarct Brain 

Liquefactive

2 What is the type of Degeneration seen in Leiomyoma 

Hyaline

3 What is the marker of acute pancreatitis

Lipase

 4 Following is not a type of Adaptations

Anaplasia

 5 Troponin I is a Marker of

Myocardial infarction 

6 Which of the following holds true for metaplasia

It is reversible

7 Metastatic calcification

Occurs in normal tissues with raised serum calcium levels

8 Pathogenesis of reversible cell injury does include the following 

Reduced Protein synthesis, Decreased ATP, intracellular lactic acidosis

9 The nature of lesion in Barrett’s oesophagus is:

Metaplastic

10 Caseous necrosis is

Conbination of coagulative and liquefactive necrosis

11 Following is  a type of Gangrene 

Dry wet, gas

12 Following is the etiological agent for gas gangrene

Clostidium welchi

 13 Following is a feature of Wet Gangrene

Common in bowel, Moist Rotten organ, Poor Prognosis

 14 Following is  a feature of Apoptosis 

Programmed Cell death, intact lysosomes, Apoptotic bodies

15 Coagulative necrosis is  seen in

 Infarct Heart, Infarct Kidney Infarct Spleen

16 Wear and tear pigment is name given to

Lipofuschin

 17 Following is  a pigment derived from haemoprotein 

haemosidderin, acid haematin, bilirubin

18 Following is the type of Extra Cellular Hyaline Deposition

Corpora Amylara

 19 Diabetic Foot is an Example of 

Wet gangrene

20 Tomb Stone is a hall mark morphologic feature of

Coagulative necrosis

 21 Fibrinoid necrosis is characterized by deposition of fibrin-like material stained by

PTAH

22 Secreatory endometrium is an example of

Hyperplasia

 23 In Atrophy the cells are 

Shrunken cells

24 Fat Necrosis occurs in

Bread

 25 Liquefactive Necrosis is seen in

Brain

 26 Common site of liquefactive necrosis is

Heart

 27 Gummatous necrosis is caused by

Treponema pallidum

28 causes of cell damage 

reduced O2 supply, genetic abnormalities, abnormal immunological reaction

29 All are features of apoptosis 

cell shrinkage, single cell shedding, chromatin condensation

30 Metastatic calcification is seen in

Hyperparathyroidism 

 31does elicit inflammatory reaction

hypoxia, toxins, antibodies

32 Steatosis is also known as 

Fatty degeneration 

33 Hyaline degeneration in rectus abdominal is muscle in typhoid feveris also known a

Zenker's degeneration

34In fatty liver due to chronic alcoholism.the following mechanism are involved 

increases fatty acid synthesis, decreased triglyceride utilization, block in lipoprotein excretion 

35Caseous necrosis is distinctive of

Tuberculosis

 36Type of necrosis occurring in myocardial infarct 

Coagulatuve

37Gangrenous organ bears blackish discolouration due to

Iron sulphide

 38“cheeselike,” necrosis 

Caseous necrosis

39Programmed and coordinated cell death is

Apoptosis

 40‘falling off’ or ‘dropping off’ is seen in 

Apoptosis 

41Free Radicals Involved in Cell Injury are 

Superoxide (O2), Hydrogen peroxide (H2O2), Hydroxyl radical (•OH

42Annexin V is used as a marker for:

Apoptosis

 43Which of the following genes is proapoptotic: 

BAX

44In atrophy, the cells are 

Shrunken cells

45decreses in size of brain with aging is example of 

Physiologic atrophy

46Wasting of muscles of limb immobilised in cast is seen in 

Pathologic atrophy

47Zenker’s degeneration of muscle seen in .

Typhoid fever

48Poliomyelitis is example of 

Neuropathic atrophy

49All are features of reversible cell injury 

Karyorrhexis, Blebs, Clumping of nuclear chromatin

50All are features of Apoptosis 

 Cell shrinkage,Single cell shedding, Chromatin condensation

51Most common cause of fatty liver is 

Alcoholism

52Cancer predesposition in vitamin A deficiency is due to

Metaplasia

2)HAEMODYNAMICS

 1Difference between hyperaemia and congestion is

 Hyperaemia is active & congestion is passive

2Most frequent cause of CVC Liver is

Right sided ventricular faillure

3Virchow's triad includes all 

Injury to vein, Venous stasis, Hypercoaguabilty of blood

4Pick the correct sequence: 

Renin-Angiotensinogen-Angiotensin I-Angiotensin II-Aldosterone

5Pulmonary hypertension is defined as systolic blood pressurein the pulmonary arterial circulation above the following cut offfigure: 

30mm hg

6Infarct Lung is

Haemorrhagic 

 7Ischemia casues injury due to 

Hypoxia, Lack of Nutrients, Accumulation of Waste Products

8Osmotic pressure exerted by the chemical constituents of the body fluids has the following features

Oncotic pressure constitutes minor portion of total osmotic pressure, Oncotic pressure of plasma is higher, Oncotic pressure of interstitial fluid is lower

9For causation of oedema by decreased osmotic pressure, the following factor is most important 

Fall in globulin level

10In septic shock, pathogenesis of endothelial cell injury involves the following mechanisms 

Interleukin-1 causes endothelial cell injury, TNF-a causes direct cytotoxicity, Adherence of PMNs to endothelium causes endothelial cell injury

11Transudate differs from exudate in having the following 

Low specific gravity, No inflammatory cells, Low protein content

12type of emboli depending upon source are . 

Cardiac emboli

13Nephritic oedema differs from nephrotic oedema in having the following 

Mild oedema, Distributed on face, eyes, Occurs in acute glomerulonephritis

14The following type of oedema is characteristically dependent odema 

Cardiac odema

15Sectioned surface of lung shows brown induration in:

CVC LUNG

 16Milroy’s disease is: 

Hereditary lymphoedema

17Pulmonary oedema appears due to elevated pulmonary hydrostatic pressure when the fluid accumulation is:

Ten fold

 18Father of cellular Pathology is 

Rudolf virchow

19Paradoxical emboli means 

20Lines of Zahn in seen in 

Thrombhs

21Fat embolism is seen in 

Pancreatitis, Diabetic Mellitus , sickle cell anemia 

22The following type of Oedema is characteristically dependent Oedema

Cardiac oedema

 23Red infarcts occurs with following 

Venous occlusion, Arterial occlusionin, in loose spongy tissues where blood can be collected in infarct zone

24 80% of systemic emboli originate from

Intracadiac mural thrombi

 25Commonest source of pulmonary embolism is 

Deep vein of leg

26Thrombi occuring in Heart chambers or Aorta are designated as

Mural thrombi

 27hypotension, weak rapid pulse,tachypnoea,cool,clammy, cyanotic skin,is representation of 

Cardiogenic shock 

28Factors responsible for oedema formation includes all 

increased hydrostatic pressure of the blood,  decreased capillary permeability, Lymphatic obstruction

 29Direct injury to the endothelium of cell causes 

Necrosis

30common derangements of body fluid are 

Oedema, Overhydration, Dehydration

31Which ONE of the following is associated with thrombosis?

Endothelial damage, .Formation of platelet aggregates., Vascular stasis.

 32CHRONIC VENOUS CONGESTION OF LIVER OCCURS IN RIGHT HEART FAILURE AND SOMETIMES DUE TO OCCLUSION OF…..

Inferior vena cava & hepatic vein

33LARGE THROMBUS IMPACTED AT THE BIFURCATION OF THE MAIN PULMONARY ARTERY IS CALLED….

Saddle embolism

. 34RISK FACTORS OF PULMONARY THROMBOEMBOLISM IS…..

Venous blood stasis

 35CAUSE / CAUSES OF FAT EMBOLISM INVOLVE….

HYPERLIPIDAEMIA, SICKEL CELL ANAEMIA, PANCREATITIS

 36THE EFFECT OF DECOMPRESSION SICKNESS DEPEND UPON…

DEPTH OF ALTITUDE REACHED, DURATION OF EXPOSURE TO ALTERED PRESSURE, RATE OF ASCENT OR DESCENT

 37TRAUMATIC CAUSES OF FAT EMBOLISM INVOLVE.....

TRAUMA TO THE BONES, TRAUMA OT THE SOFT TISSUES

 38EXAMPLES OF LIQUID EMBOLISM ARE ALL 

FAT GLOBULES, AMNIOTIC FLUID, BONE MARROW

.. 39AIR EMBOLISM IS SEEN COMMONLY IN VEINS OF WHICH SITE?

Head and neck

 40VIRCHOW DESCRIBED THREE PRIMARY EVENTS WHICH PREDISPOSE TO THROMBUS FORMATION:

ENDOTHELIAL INJURY, ALTERED BLOOD FLOW, HYPERCOAGULAB ILITY OF BLOOD

41INJURY TO VESSEL WALL CAUSES… 

Vasoconstriction of small blood vessel

42…….SYNTHESIZED BY THE ENDOTHELIAL CELLS BINDS TO GPIB AND FORMS A FIRM ADHESION OF PLATELETS WITH ECM

Von willebrands factor

 43SEQUENCE OF EVENTS OF ROLE OF PLATELETS IN THROMBOSIS IS …….

PLATELET ADHESION, PLATELET RELEASE REACTION, PLATELET AGGREGATION

 44DEFICIENCY OF GPIB AND VWF WOULD RESULT IN…. 

DEFECTIVE PLATELET ADHESION, ABNORMAL BLEEDING

45STABLE HAEMOSTATIC PLUG IS FORMED BY THE ACTION OF…

FIBRIN, THROMBIN, THROMBOXANE A2

 46THE THROMBU SMAY ENLARGE IN SIZE DUE TO MORE AND MORE DEPOSITION FROM THE CONSTITUENTS OF FLOWING BLOOD . THIS PROCESS IS KNOWN AS 

Propagation

47ARTERIAL THROMBI ARE FORMED FROM…

Endothelial cell injury

 48THE LINES OF ZAHN ARE FORMED FROM LAYER OF LIGHT-STAINING AGGREGATED PLATELETS ADMIXED WITH….

Fibrin meshwork and dark staining layer of red cells

 49MAIN TYPES OF OEDEMA

Localised and general 

50Lines of ZAHN is a chararacteristic appearance of laminations in thrombi when formed in 

aorta

51All infarcts are associated with coagulative necrosis 

Kidney, Spleen, Liver

52The minimal blood loss that causes hypovalemic shock is

10-15%

 53Cancer predesposition in vitamin A deficiency is dueto

Metaplasia

3)INFLAMMATION, WOUND HEALING & REPAIR

 1The immediate vascular response in Acute Inflammation is

 Vasoconstriction 

2The cardinal signs of Inflammation are all 

Rubor, Calor, Dolor

3Increase in vascular permeability is mainly due to

Endothelial damage

 4Neovascularisation is the term used for 

New capillary formation

5 6The process of escaping of neutrophils out into the extravascular space is termed as

Emigration

 7Diapedesis is a passive process of escape of 

Erythrocytes, RBC

8The transmigration of leucocytes after crossing several barriers to reach the interstitial tissues is called

 Chemotaxis

9All are the stages of Phagocytosis 

Recognition and Attatchment, Engulfment, Killing and degradation 

10Reed Sternberg cell is a giant cell seen in 

Hodkin"s lymphoma

11The cell derived from B lymphocytes with an eccentric nucleus having cartwheel arrangement of chromatin is

Plasma cell

 12Pseudomembranous inflammation is seen in infection with

Corynebacterium diphtheriae

 13Diffuse Inflammation of the soft tissues resulting from spreading effects of substances like hyaluronidase released by some bacteria is termed

Cellulitis

 14Systemic effects of acute Inflammation includes all 

Fever,Gonorrhea, diptheria

 15Granuloma formation occurs in 

Tuberculosis

16The cells which continue to multiply throughout life under normal physiological conditions are

Epithelial cell of epidermis 

17The cells which lose their ability to proliferate after adolescence are

Cardiac muscle cell

18Regeneration is when healing takes place by proliferation of 

Parenchymal cells

19All are features of Granulation tissue 

Granular and pink, Proliferated new vessel, Fibroblasts & collagen 

20Healing by first intention has all the stages 

Initial haemorrhage, Acute inflammatory response, Epithelial changes

 21Wound contraction begins on 2nd or 3rd day and is completed in

2weeks

 22Local factors that delay the process of healing are all 

Infection, Poor blood supply, Foreign bodies 

23The fracture communicating to skin surface is 

Compund

24The Giant cells in a tubercle are

Langhan's 

 25Healing by first intention occurs in al wounds 

Clean & Uninfected, Surgically incised, Edges approximated with sutures

 26Main cytokines acting as mediators of inflammation one as under 

Interlukin-1(IL-1), Tumour necrosis Factor α (TNF-α), Interferon ϒ (IFR- ϒ) 

27Transudate differs from Exudate in having following 

low inflammatory cells, low protein content, low specific gravity 

28Histological hall mark of chronic inflammation are all 

mononuclear infilterate, fibroblast proliferation, fibrosis 

29Mast cells produce 

Histamin

30Transudate has all of following characters 

specific gravity is 1.006, clear & transperant, protein contains less than 3gms 

31Repair process is enhanced by all 

Immobilisaition, non infected wound, middle age 

32During acute inflammation the movement of RBCs through the gaps between the endothelial cells is called as 

Diapedesis

33All are example of plasma derived mediators of acute inflammation 

kinnin system, comolement, clotting system 

344 cardinals signs of acute inflammation were given by 

Celus

35The cell that predominates during 1 TO 12hrs after onset of of inflammation

Neutrophils

 36All are the features of secondary healing that differentiates it from primary healing 

inflammatory reaction is more intense, larger granulation tissue, wound contraction

37Exudate has all of following features 

Turbid on gross, specific gravity 1.018, increased capillary permeability is the cause 

38IN CARDINAL SIGNS OF INFLAMTION PAIN MEANS

Dolor

 39Flare is the bright reddish appearance due to

Vasodilation 

40Histamine - It is stored in the granules of 

Mast cell, basophil, plaletes

41PLASMA-DERIVED MEDIATORS are 

The kinin system, The clotting system, The complement system 

42The major proinflammatory actions of these neuropeptides are all 

Increased vascular permeability. Transmission of pain stimuli, Mast cell degranulation.

43 Prostaglandins PGE2 act on blood vessels to causes

Vasodilation and bronchodilation

44Healing is example of

Neovascularisation

 45Which of the is the earliest event that occurs in the vascular flow during acute inflammation

Vascontriction

 46All the following are effects of bradykinin 

Increase vascular permeability, smooth muscle contraction pain

47All the following are mononuclear cells 

Macrophaves, lymphocytes, plasma cell

48The histological hall marks chronic inflammation are all 

Mononuclear infiltrate, fibroblast proliferation, fibrosis

49Epitheloid cells in granulation are modified 

Macrophages

50The epitheliod cell is adapted for

Extracellular secreation

4)HEMATOLOGY

 1Reedsternberg joint cell are seen in

Hodgekin's disease

 2Hypochromic microcytic anaemia is seen in

Iron deficiency anemia 

3Heinz bodies are seen in 

G6PD Deficiency 

4Haemophilia A is caused due to defeciency of

Factor 8

 5In sickle cell anaemia point mutation occur at 

12th position of betachain

6Prothrombin time is prolonged in all 

liver disease, Vit K deficiency, Disseminated intravascular coagulation 

7Philadelphia chromosome is characteristic of

Chronic myeloid leukemia 

8Macrocytic megaloblastic Anaemia is cardinal feature of defeciency of 

Vit. 12

9The following disorders may result from plasma loss in dehydration from vomiting

Relative /spurious polycuthaemia

 10Increased osmotic fragility of red cell is seen in

Sherocytosis

 11Lynphocytosis is seen in

Tuberculosis

 12Leucopenia is characteristic of

Enteric fever

 13Basophils are increased in

Chronic myeloid leukemia

14Direct coomb's test is done with the patients 

RBC's

15Haemolytic anaemia that appears right from birth is

Hereditary spherocytosis

16Classic haemophilia is due to deficiency of 

Factor 8

17In sickle cell anaemia point mutation occur at 

6th position of alpha chain

18Prothrombin time is prolonged in all 

Liver disease, vit K deficiency, obstructive jaundice 

19Pernicious anaemia is caused by deficiency of 

Intrinsic factor

20subacute combined degeneration of spinal cord is seen in deficiency of 

Vit B12

21All are Autosomal Recessive Inheritance diseases 

thalassaemia, Sickle cell anaemia, Wilson’s disease

 22In sickle cell anemia RBC life span is 

20dags

23Hemorrhagic diathesis become clinically evident when platelet count fall to

20,000

 24Which of following condition indicate thrombocytopena

Normal clotting time + prolong bleeding time

 25In Polycythemia vera erythropoietin levels

Decrease

 26Iron deposition into parenchynal cells of liver is characetristic feature of

Idiopathic Hemosiderosis

5)MALNUTRITION 

1Important method to guage Obesity is

BMI index

 2Correct Marker of Starvation is 

Excessive production of ketone bodies

3Protein energy or Protein calorie malnutrition occur in case of

Kwarshiorkar

 4Overall calcium deficiency reflects as

Osteoporosis 

 5Which is water soluble Vitamin

Vit c, Vit B, Biotine

6Chronic cunsumtion of Large Doses of Vit A produces 

Sever headache with stupor

8)Name of Vit D3 is 

Cholecalciferol 

9)Which is  effect of Obesity

Hyperinsulinaemia, atherosclerosis, hypertension 

10)The main physiological function of Vit K is

Carboxylation of coagulation factor no II

11Primary cause of Malnutrition syndrome is 

Coeliac disease 

12A child is considered to have Maramus when weight level falls to of normal for sex,Height,and age 

60%

13Generalised dependent Oedema is seen in

 Kwashiorkar

14Severe form of Protein Energy Malnutrition is called

Cachexia

 15Defeciency of Vitamin D causes 

Rickets

16Bitot’s spots seen in

Vitamin A Deficiency 

17Wernicke-Korsakoff’s syndrome 

Vitamin B1 Deficiency  

18Dermatitis, Diarrhoea, Dementia IS TRAID OF

Pellagra

 19Vitamin D deficiency in adult is

Osteomalacia

 20Rachitic rosary SEEN IN 

Rickets

21Pigeon-chest deformity In ricket due to

Anterior Protrusion of sternum 

 22Which vitamins is required for collagen synthesis

Vit. C

6)IMMUNITY

 1Arthus reaction is what type of hypersensitivity reaction 

Localised immune complex

2Function of humoral immunity is 

Antigen exposure

3The process induced from the antigen antibody reactions in vitro are

Agglutination, neutralisation, complement fixation 

4Histologically, the characteristic lesion in SYSTEMIC LUPUS ERYTHEMATOSUS(SLE) is

Fibrinoid necrosis

 : 5Tuberculin test is an example of which type of hypersensitivity reaction 

Type IV

6Herd Immunity is best defined as immunity developed in how much percentage of population against the infective agent

0.8%

 7Hapten is derieved from the greek "haptein" which means 

To fasten

8 is an example of precipitation

VDRL test

 9Cross linking of mast cell bound IgE by the allergen is seen in which hypersensitivity reaction

Type I

 10Internal Society of Blood Group has identified 

20blood group and 7antigen

11Deposition of antigen antibody complexes is characteristic of hypersensitivity

Type 3

 12The macrophage originate from 

Monocytes

=13 are part of innate imunity 

NK cells, Dendrites cells, neutrophils

14 antibodies are hallmark of type I hypersensitivity

IigE

 15Transplant antigens (mhc complex) are located on chromosome 

6

16The induction and regulation of immune response are mediated by short acting soluble mediators called 

Cytokines

17B cell defects are

Bruton's X-linked aggamaglobulinaemia, IgA deficiency, Autosomal recessive aggamaglobulinaemia

18common derangements of body fluid are

Oedema, ovdrhydration, dehydration 

19Lardaceous amyloidosis is seen in

Spleen

 20The cells which contains cytoplasmic granules ,initiates inflammation are

Mast cell

 22The immunity plays a vital role in preventing epidemic diseases is.... 

Herd immunity 

23Antibody which can cross the placental barrier is...

IgG

 24Antibody that take part in type I hypersensitivity reaction is...

Ig E

 25The level of antibody increases in parasitic infection is

 Ig E

26The type of hypersensitivity reaction in graves disease is

Type 2

 27The antibody which shows dimetric form is....

Ig A

 28the self tissues transfered from one part of body site to another, in same individual known as 

Autograft

29Tissue transferred between genetically identical individuals

Isograft

 30Tissue transferred between genetically nonidentical members of same species

Allograft

 31Tissue transferred between different species

Xenograft

7)AMYLOIDOSIS 

1WHICH OF THE FOLLOWING ARE THE TEST FOR AMYLOIDOSIS?

 Biopsy, congo red test

2AMYLOID IS DEPOSITED IN WHICH PART OF THE KIDNEY?

Glomeruli

3ARRYTHMIA IS PRODUCED DUE TO… 

Deposition of amylod in conctive systems 

4THE DIAGNOSIS OF AMYLOID DISEASE CAN BE MADE FROM THE FOLLOWING INVESTIGATIONS ,

In vivo congo red test, biopsy examination, protein electrophoresis 

5IN SYSTEMIC AMYLOIDOSIS WHICH BIOPSY PROVIDES THE BEST DETECTION RATE? 

Rensl biopsy

6WHICH IS THE LOCATION OF AMYLOID DEPOSIT? 

Parenchyamal cell, inthe extra cellular matrix

*7WHICH OF THE FOLLOWING CONFIRMS THE PRESENCE OF AMYLOID? 

Apple green birefringence

8AMYLOID DEPOSITION IN THE SPLEEN MAY HAVE FOLLOWING PATTERN?

Sago spleen, lardaceous spleen

9WHICH MAY BE THE SITE FOR TUMOUR FORMING AMYLOID? 

Tongue

10AL PROTEIN IS PRODUCED BY

Immunoglobulin Secretary cells

11SAA STANDS FOR 

Serum amyloid associated proteins 

12IDENTIFY  FIBRILAR PROTEIN

 TRANSTHYRETIN, AMYLOID BETA PEPTIDEA, BETA_2 MICROGLOBULIN

13WHICH VARIETY OF AMYLOID PROTEIN WAS ISOLATED FIRST 

Apolipoprotein E

14ARRANGE PROPER SEQUENTIAL EVENTS IN AMYLOIDOSIS :

 i) POOL OF AMYLOGENIC PRECURSOR 

iii) A NIDUS FOR FIBRILOGENESIS

ii) PROTEOLYSIS  

iv) NON FIBRILAR COMPONENETS

 15WHICH COMPONENT PLAYS IMPORTANT FUNCTION IN FOLDING AND AGGREGATION OF FIBRILAR PROTEINS

AP, AA

16WHICH OF THE FOLLOWING IS  INCLUDED IN LOCALISED AMYLOIDOSIS

17HEREDOFAMILIAL AMYLOIDOSIS IS SEEN IN

Haemodylasis associated amylodosis

 18PATTERN 2 OF AMYLODOSIS BASED ON CLINICAL LOCATION INVOLVES

After

 19WHICH DISEASE IS ASSOCIATED WITH SENILE CEREBRAL AMYLODOSIS  

Transmissiblr encephalopathy 

20Sago spleen is seen in 

Haemochromatosis

21Extracellular deposits of fibrillar proteins responsible for tissue damage and functional compromise is seen in 

Amyloidosis

22The immunity chiefly responsible for granuloma formation is

Cell mediated immunity

23All of the following are permanent cells 

Nerves, skeletal-muscle, cardiac muscle cells

24A positive Mantoux test indicates that the child 

Is suffering from active TB

 Has had BCG vaccination recently 

Has had TB infection

*25Coomb's test is

An antiglobulin test

 26Ig E is increased in infections which are

Allergic

 27Immunity is life long following

Measles 

 28The earliest Immunoglobulin synthesized by foetus is

IgM

 29Truth about BCG is 

Is produced by repeated subculture 

8)NEOPLASIA 

1Tumour containing all three germ layers is called.

Teratoma

 2Lack of differentiation is called

Anaplasia

3Which of the following is known as the 'Guardian of the Genome' 

P53

4True statements about p53 gene are all 

Arrest cell cycle at G1 Phase, Product is 53 KD Protein., Located on Chromosome 17.

 5Alpha-fetoprotein is detected in case of

Hepatocellular carcinoma 

6Malignant tumours of epitnelial origin are called

Carcinoma 

7TNM staging was developed by 

UICC

8Benign tumours are

Encapsulated, well circumscribed 

9Rhabdomyoma is tumour of

Skeletal muscle

 10Common sites of Blood borne metastases are as follows 

Spleen

11Enzyme which prevents aging 

Telomerase

12Spread of malignant cell from one site to the distant site is known as 

Metastasis 

13The organ to which metastasis never occur is

Spleen

 14Features of benign tumours are all 

Slow growth, capsulated, absence of metastasis

15Cytological criteria for malignant tumor includes all 

Hyperchromatism, capsule, increased nuclear cytoplasmic ratio

16Lipoma is which type of tumor 17All the following are indicative of anaplasia except 18Lymphnode enlargement in proximity of cancer is due to 19The criteria in staging of tumor include all except 20Most common islet cell tumor is 21Most common tumor of urinary bladder is 22Most common tumor among women 23Most common benign tumor of female breast is